VIAGRA New Study – Does It Really Reduce Mortality


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25 responses to “VIAGRA New Study – Does It Really Reduce Mortality”

  1. OMAD 6 years: https://www.youtube.com/watch?v=QCP2nHXO1Bc

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  2. Correlation studies are worthless.
    And the ones looking at one isolated molecule or one single biochemical mechanism are also worthless.
    The channel Biolayne also exposes studies and claims like that all the time

    • @Siim Land Don’t forget the potential common side effect of blue-vision with Sildenafil use. Some reports of it being irreversible. I don’t think you see that in Tadalafil though.

    • @Siim Land Invalid studies don’t magically become valid just because there are many of them. And even if the studies are valid, it certainly isn’t valid to make assumptions about causal mechanisms. Was it the increase in blood flow reduced mortality? Or was it the reduction in stress for those men who can now have sex again, and aren’t chronically embarrassed and stressed about impotence? Or was it the fact that men taking viagra are sexually active, and therefore presumably have better, healthier lives than men who don’t take viagra because they have no occasion to get it up for anyway? Likely it’s a combination of many factors. So let’s not jump to conclusions and start popping boner pills hoping to eek out an extra 5 years of life.

    • No, they are not worthless. Correlation studies give us hints. Most of the mortality studies on exercise are correlational. Were they worthless? Nope, they gave us pretty good data that moderate to high amounts of exercise are generally good for you.

      It is typically impractical to run RCTs for years to determine mortality. So if you want studies on mortality, they’re mostly going to be observational. Furthermore, one can attempt to adjust for co-founding factors (income, education, marriage status, hypertension, cvd, diet quality, shift work, sleep etc . . ). Is it perfect? No. Is it worthless? Also, no.

    • @jack buaer Sometimes yes, sometimes no. Again, the only way to distinguish the “hints” from the noise is with controlled studies. I understand that controlled studies are very difficult to accomplish, especially for humans. But a lack of solid causal evidence doesn’t make correlation the new causation by default.

  3. The issue here is if used for heart health (not for ED) what should be the daily dosage? Like 5mg or 10mg daily? The issue is that Viagra has an active action for just about 4 hours so I think to maintain regular blood levels of the drug, it needs to be taken 3 or 4 times a day at regular intervals.. curious about this..

  4. The problem with correlation studies is that it’s practically impossible to extrapolate the mechanisms behind the outcomes. For instance, you’re quick to chalk the benefits of viagra up to increased blood flow, but I think it’s equally likely, if not more so, to be the reduction in stress caused by treating ED, a condition imagine is extremely stressful and embarrassing. Not to mention the fact that in a population of men with ED, those taking viagra are almost certainly doing so because they are sexually active. So it’s just as likely to be the fact that these men have spouses, families, somebody to be intimate with, and/or better social lives, and likely less stressful and lonely lives than those who don’t take viagra because they presumably have no need to pitch a tent.

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